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However, the therapeutic utility of doxorubicin is undermined by its dose-dependent cardiotoxicity, possibly arising as a result of futile redox cycling, with NADH dehydrogenase within the mitochondria proposed as the most likely site of anthracycline reduction 9,10

By protecting the mitochondria from lipid peroxidation, this synergistic formula helps restore the body's ability to generate and utilize ATP efficiently

Nonetheless, a detailed understanding of subcellular NADP redox dynamics is limited by the availability of appropriate tools

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Gynecologic Oncol (2006) 103(2):6927

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