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Lu, Sheen, and Shee 2013

Furthermore, various studies have explored strategies to enhance neurological functions in T21 mice through the promotion of neurogenesis

By disrupting microtubule networks, cellular function and axonal transport is compromised, such as the aforementioned mitochondrial transport deficits [116]

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Our earlier reports indicate that loss of hepatocyte beta-catenin protects mice from experimental porphyrin-induced liver injury